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Immunology

Infection with one dengue serotype confers long- lasting type-specific protection, a benefit that may be more apparent than real because the type-specific protection also brings a greatly increased susceptibil­ity to severe dengue disease in the event of infection with dengue virus of another serotype.

Epidemiolo­gical studies clearly link the severe forms of dengue disease to previous dengue infection and to trans­placental acquisition of maternal dengue antibody.

Dengue virus infects mononuclear phagocytes, and there is both in vitro and in vivo evidence that the interaction is facilitated and severe infection more likely when antibody to another dengue sero­type is present. This phenomenon alone has impor­tant implications for the development of dengue vac­cines. Administration of monovalent or polyvalent vaccines, even if they were now available for human use, carries with it serious risk of predisposing par­tially immune populations to further and more seri­ous dengue virus disease.

Secondary dengue infections that carry no risk of dengue hemorrhagic fever or dengue shock syn­drome can occur when sequential dengue infections occur at intervals of 5 or more years, when sequen­tial infections do not end with dengue type 2, and when two or more dengue viruses, including type 2, are endemic in the same area. Malnourished chil­dren and alpha-thalassemia patients have a greatly reduced risk of dengue hemorrhagic fever and dengue shock syndrome.

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Source: Kiple Kenneth F. (Editor). The Cambridge World History of Human Disease. Cambridge University Press,1993. — 1200 p.. 1993

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